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Autism spectrum disorder (ASD) is a complex neurodevelopmental condition marked by language delays, social-interaction difficulties, repetitive behaviors, and a range of cognitive challenges.
Light can strongly influence mood and behavior. Color tone and brightness affect most people, and individuals with autism may respond even more intensely.
Children with ASD often show heightened sensitivity to lighting. Soft hues such as pale blue can promote calm and creativity, whereas flickering, humming, or harsh lights may cause discomfort or agitation. Dimmable, quiet fixtures give caregivers better control over the sensory environment.
Natural light also matters. Reduced daylight in winter can contribute to seasonal affective disorder (SAD), sometimes worsening irritability or low mood. Access to daylight or a daylight-mimicking source may ease these effects.
Bright-light sessions may also help regulate circadian rhythms—the internal "clock" that governs sleep–wake cycles. A short morning session (starting with as little as 10–15 min and building up to about 30 min) can support easier sleep onset at night and greater daytime alertness. Evening exposure is usually discouraged because it might delay sleep.
This information is educational and not a substitute for professional medical advice.
Further reading:
A study examined whether low-level laser therapy, a form of photobiomodulation, could ease irritability linked to autistic spectrum disorder in children and adolescents aged 5–17 years. Twenty-one of 40 participants received eight 5-min procedures at the base of the skull and temporal areas over 4 weeks (active group). All were assessed with the Aberrant Behavior Checklist (ABC)—global score and five subscales—and the Clinical Global Impressions (CGI) Scale at baseline, week 2, week 4, and week 8. The adjusted mean change in ABC irritability subscale from baseline to endpoint was –15.17 points greater in the active group (ANCOVA F = 99.34, p < 0.0001). The authors suggest low-level laser therapy may reduce irritability and related behaviors, with improvements persisting and slightly increasing after treatment ended.
Previous work indicates that low-level laser light can traverse the skull. The proposed mechanism involves quantum optical induced transparency, which may enhance light transmission through dense tissue, allowing non-invasive irradiation of deeper brain structures.
The data suggest photobiomodulation produced a statistically significant and clinically meaningful improvement in core autism-related behaviors in this cohort, with gains continuing for up to six months after the final session.
This information is educational and not a substitute for professional medical advice.
LLLT may produce a therapeutic effect by delivering non-ionizing light—such as lasers, light-emitting diodes, or broadband sources—in the visible red (600–700 nm) and near-infrared (780–1100 nm) ranges. This non-thermal process begins when chromophore molecules absorb light of the appropriate wavelength. Chromophores give color to compounds like hemoglobin, myoglobin, and cytochromes; after absorbing a photon, an electron moves to an excited state. The resulting physiologic changes appear to occur when photons displace the inhibitory signaling molecule nitric oxide (NO) from cytochrome c oxidase, which may increase electron transport, mitochondrial membrane potential, and the production of ATP, NADH, RNA, and other mitochondrial products. The leading hypothesis is that this release of NO from the enzyme enhances electron flow, mitochondrial membrane potential, and ATP synthesis.
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Further reading: PubMed database
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